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Smith D.C., Kalebic T., Infante J.R., Siu L.L., Sullivan D., Vlahovic G., Kauh J.S., Gao F., Berger A.J., Tirrell S., et al. Phase 1 study of ixazomib, an investigational proteasome inhibitor, in advanced non-hematologic malignancies. Radhakrishnan S.K., den Besten W., Deshaies R.J. p97-dependent retrotranslocation and proteolytic processing govern formation of active Nrf1 upon proteasome inhibition.
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Our current study supports the use of BET inhibitors in conjunction with proteasome inhibitors. Interestingly, treatment with either BET inhibitor alone did not show significant changes in the UPR genes (with one exception in MDA-MB-231 where ATF3 mRNA increased approximately four-fold in response to JQ1/I-BET763). Together, our results indicate that BET inhibitors have the ability to aggravate the UPR initiated by proteasome inhibition. This notion was further reinforced when we examined CHOP and BiP at the protein level.
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Darktrace targets £3.8bn valuation as it picks banks for London float – Sky News
Darktrace targets £3.8bn valuation as it picks banks for London float.
Posted: Fri, 06 Nov 2020 08:00:00 GMT [source]
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We and others have previously established Nrf1 as a master transcription factor of the proteasome genes . In response to proteasome inhibition, Nrf1 is activated resulting in de novo synthesis of proteasome genes leading to a bounce-back response or recovery of proteasome activity . Here, using three different cancer cell lines, we investigated the changes in proteasome gene transcription in response to CFZ and BET inhibitors JQ1, and I-BET762.
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